piše na internetu da sadrži K12.
Nadam se da to nije ovaj K12:
K-12

A strain was isolated from a stool sample of a patient convalescent from diphtheria and was labelled K-12 (not an antigen) in 1922 at Stanford University.[9] This isolate was used in 1940s by Charles E. Clifton to study nitrogen metabolism who deposited it in ATCC (strain ATCC 10798) and lent it to Edward Tatum for his tryptophan biosynthesis experiments,[10] despite its idiosyncrasies due to the F+ λ+ phenotype[7] In the course of the passages it lost its O antigen[7] and in 1953 was cured first of its lambda phage (strain W1485 by UV by Joshua Lederber and colleagues) and then in 1985 of the F plasmid by acridine orange curing by G Plunkett, M. Berlyn, M. Guyer MG1655). Strains derived from MG1655 include DH1, parent of DH5α and in turn of DH10β (rebranded as TOP10 by Invitrogen[11]).[12] An alternative lineage from W1485 is that of W2637 (which contains an inversion rrnD-rrnE), which in turn resulted in W3110.[8] Due to the lack of specific record-keeping, the "pedigree" of strains was not available and had to be inferred by consulting lab-book and records in order to set up the Harvard Stock Centre.[9] The different strains have been derived through treating E.coli K-12 with agents such as nitrogen mustard, ultra-violet radiation, X-ray etc. A full list of Escherichia Coli K-12 strain derivatives and their individual construction, genotypes, phenotypes, plasmids and phage information can be viewed at Ecoliwiki.
Također piše da sadrži ksilitol, koj mi se čini OK. Evo što wikipedia kaže za ksilitol:
Xylitol is a “tooth-friendly”, nonfermentable sugar alcohol.[12][13] A review of the scientific literature[14] on the efficacy of xylitol has indicated dental health benefits in caries prevention, showing superior performance to other polyols (polyalcohols). This is because the structure of xylitol contains a tridentate ligand, (H-C-OH)3 that can rearrange with polyvalent cations like Ca++. This interaction allows for Ca++ to be transported through the gut wall barrier and through saliva may remineralize enamel before dental caries form.[15]
Early studies from Finland in the 1970s found that a group chewing sucrose gum had 2.92 decayed, missing, or filled (dmf) teeth compared to 1.04 in the group chewing xylitol gums.[16] Recent research[17] confirms a plaque-reducing effect and suggests the compound, having some chemical properties similar to sucrose, attracts and then "starves" harmful micro-organisms, allowing the mouth to remineralize damaged teeth with less interruption. (However, this same effect also interferes with yeast micro-organisms and others, so xylitol is inappropriate for making yeast-based bread, for instance.) This is because cariogenic bacteria prefer fermentable six-carbon sugars, or disaccharides such as sucrose, as opposed to the nonfermentable xylitol, whose antimicrobial properties then "starve" the bacteria, reducing their growth and reproduction.[15] Most of these studies suggest that at least 6 grams of xylitol per day is needed for dental efficacy; for most chewing gum or breath mints this would require 12 pieces per day (op cit).
Xylitol is specific in its inhibition of the mutans streptococci group, bacteria that are significant contributors to tooth decay.[18] Xylitol inhibits mutans streptococci in the presence of other sugars, with the exception of fructose. Daily doses of xylitol below 3.44 grams are ineffective, and doses above 10.32 grams show no additional benefit.[19]
Saliva containing xylitol is more alkaline than saliva which contains other sugar products. After taking xylitol products, the concentration of basic amino acid in saliva may rise. When saliva is alkaline (i.e., its pH is above 7), calcium and phosphate salts in saliva start to precipitate into those parts of enamel where they are lacking.[6]
Xylitol also inhibits the growth of Streptococcus pneumoniae, as well as the attachment of Haemophilus influenzae on the nasopharyngeal cells, making xylitol nose spray a very marketable product.[15]
However, despite these promising conjectures two systematic reviews of clinical trials could not find conclusive evidence that Xylitol was indeed superior to other polyols, i.e. Sorbitol (Mickenautsch and Yengopal, 2012) or equal to that of topical fluoride in its anti-caries effect (Mickenautsch and Yengopal, 2012b).
Xylitol-based products are allowed by the U.S. Food and Drug Administration to make the medical claim that they do not promote dental cavities.